NAD+ is an essential molecule in biological processes such as metabolism and lifespan. Deficiencies in NAD+ lead to complications in the model organism C. elegans. We research a phenotype caused by a gene-environment interaction involving the riboside pathway of NAD+ synthesis. This delayed hatching phenotype occurs when nmrk-1 is knocked down and the animal is grown in oxidative stress environments. We seek to understand the contribution of environmental factors to the expression of delayed hatching.