Neurons are exposed to a variety of stressors, resulting in axon injury and neurodegenerative disease. Using Drosophila as a model, we aim to better understand how neurons respond to axon injury, which could further knowledge of these disease mechanisms and aid in treatment development. Chordotonal neurons, a ciliated sensory cell, must regenerate axons differently than dendritic arborization neurons. Wnt molecules secreted by epithelial cells are potential ligands that initiate γtubulin localization at dendritic branchpoints.