Neurons, which transmit signals throughout the body, can be injured in many ways, and must regenerate after injury. Using a deficiency screen in drosophila, we have identified two lines: one causes decreased regeneration after injury, and the other shows abnormal dendrite morphology before injury. Experiments revealed that the protein skeletor functions in dendrite regeneration. The system of injury that has been used is inefficient, so we also propose a new injury method for the dendrites.