Hormonal contraceptives (HC) act on hormone receptors in the hypothalamus and diminish production of ovarian hormones like estrogen and progesterone via negative feedback regulation of the HPG axis. Here, we examined adolescent and young adult binge drinking during contraceptive hormone treatment. Adolescent female C57BL/6J mice were given 10% sucrose solution for one week beginning on PND 36 for acclimation. From PND 43-77, the control group (n=6) received 250µL of 10% sucrose solution (SUC), and the experimental group (HC, n=6) received 10% sucrose with a suspended combination dose of 0.075 µg/ml ethinyl estradiol and 3 µ/mL levonorgestrel. These mice also consumed alcohol using Drinking in the Dark (DID) model for binge drinking from PND 50-77. In this model, mice are given intermittent access to 20% EtOH in water 3 hours into the dark cycle (lights off). Interestingly, we found a decrease in total ethanol (EtOH) consumption in mice that were administered HC compared to SUC (p=0.0383). To understand this change in behavior better, it is necessary to explore how our HC model affects ovarian biology. Future work will identify mechanisms through which HC reduces alcohol consumption by evaluating ovarian function, testing for estrous acyclicity and inhibition of ovulation.